Good find KK2.

For the Drs and scientists here, does the mitochdrial calcium transport process they talk about in the article have anything to do with alkaline phosphotase that apabetalone seems to reduce?

My thought here is with regard to the last paragraph of the article; "Problems with calcium regulation and metabolic dysfunction have been associated with Alzheimer's development and malfunctioning neurons for some time. Intracellular calcium signalling plays an essential role in the synaptic transmission and Brian cell communication".

We know from previous trials that apabetalone reduces alkaline phosphotase, is this one in the same thing?

tada